Monday, April 29, 2013

EFSA: Risk communication

http://www.efsa.europa.eu/en/efsawhat/riskcommunication.htm


Sent from my iPad

EFSA: Videos

http://www.efsa.europa.eu/en/news/videos.htm


Sent from my iPhone

Updates of Mind Maps

Thanks to the students who reminded me that I have not uploaded some of the lecture Mind Maps. I have checked and upload the missing ones. If you find that I still miss some, please let me know.

HS Kwan

Review session questions



Question: why is the most heat-sensitive enzyme inactivated determining the growth and servival of the microorganism.

Answer: Refers only to essential enzymes. Inactivating the most hear-sensitive essential enzyme would stop the growth and may affect the servival of the cells.






 Question: How does Prp changed into Prp-sc  and cause mad cow disease?

Answer: Prp may change into Prp-sc by mutation. OR human acquired Prp-sc from food source. Prp-sc can recruit normal Prp and change its form to Prp-sc. Prp-sc aggregates to form rod-shaped polymers that can block the nutrient and energy supply of brain cells and cause mad-cow disease.





Review Session 20130429 Question from students and Answers 5

Question:  
  •  Any more information on DAEC?

Not much is known about this bacteria
  •  Fungal toxin included in examination?
 Not covered in lecture--> not included in examination
  • Food preservation covered in examination?
No direct coverage in lecture. But still need to note things like Nitrate effect on Redox--> effect on anaerobic bacteria growth 

Review Session 20130429 Question from students and Answers 4

Question:

Are all Clostridium spp unable to ferment sugar?

Consult: Microbe Wiki http://microbewiki.kenyon.edu/index.php/Clostridium_perfringens

C. perfringens has all the glycolytic genes and should be able to ferment sugar.

C. botulinum--no report on sugar fermentation

Review Session 20130429 Question from students and Answers 3

Question: Shigella


  • Does it infect other primates?
Yes.  
  • Pathogenicity picture explain more?

Note the picture:




  • Endocytosis- Shigella gets into enterocyte
  • Enclosed by vacuole
  • Break out from vacuole
  • Propagate in cell
  • Cell secretes cytokines
  • Cytokines attract PMN
  • PMN opens up tight junctions between enterocytes
  • Damage enterocyte layer
  • Shiga toxin from Shigella gets into blood
  • Damage blood vessels
  • Blood diarrhea and systemic symptoms

Not in exact sequence, e.g., shiga toxin can be produced any time